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Several studies show that LPS promotes the aggregation of peripheral amyloid, tau, and α-synuclein, thereby accelerating fibrillogenesis (Lee et al., 2008

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Peter handles this question with empathy, because these stories are sincere and he hears them constantly He’s hearing them both directly and indirectly from patients who are sharing stories of their friends or family members But a testimonial describes what happened after someone took a drug It can’t tell you what would’ve happened without it It also fails to often reflect what else was being taken or done with that drug And those counterfactuals are the whole ballgame Start with the biology of injuries Musculoskeletal injuries tend to improve on their own, and they fluctuate a lot People almost always start a peptide when they’re at their worst—which is exactly when you’d expect things to get better anyway, drug or no drug That’s simply regression to the mean This is a well-documented phenomenon in human physiology Then, layer on everything else people are doing at the same time They’re probably resting, modifying activity, doing physical therapy, perhaps taking anti-inflammatory drugs, sleeping better, eating better, training smarter, and perhaps even taking anabolic agents “ BPC-157, CJC-1295, or some other peptide stack, is just one part of a sea of variables, and yet it always seems to be the thing that gets the credit .”‒ Peter Attia One brief example Peter’s friend who wanted to start peptides said another friend of his was taking it and it was having a remarkable effect He shared the pre and post photos of his friend before and after he was on his peptide stack And there is no denying these photos, there was an enormous improvement Peter replied, “ This looks amazing