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Its urate-lowering effect was not accompanied by detectable suppression of hepatic urate production, but was associated with renal and intestinal urate-handling-related responses
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Especially under ischemia‒reperfusion conditions, reducing the level of acylcarnitine has a significant effect on the energy metabolism of the heart and the maturity and proliferation of cardiomyocytes because reducing the utilization of fatty acids and increasing the reliance on glucose oxidation as an energy source can promote the recovery of myocardial function at a lower rate of oxygen consumption
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