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It attaches to sulfhydryl proteins, interrupts the synthesis of structural proteins, modifies homeostasis of calcium, heightens lipid peroxidation, decrease saturated fatty acid, and increases cell membrane fatty acid contents [13] ( Figure 1 )

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TLR4, for example, activates intracellular signaling via the MyD88 adaptor protein, leading to NF-κB activation and subsequent transcription of pro-inflammatory cytokines (TNF-α, IL-1β, IL-6), chemokines (CCL2, CXCL10), and other mediators [444]