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NR3C1 is located at the apical level of fully differentiated epithelial villi, and it controls cldn-1 transcription via binding to glucocorticoid response elements (GREs) on the CLDN1 promoter at its transcription start site (TSS)
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493 Emerging evidence also suggests that sodium chloride modulates T cell exhaustion by enhancing TCR signaling, metabolic fitness, and cytotoxicity, 494,495 suggesting that sodium chloride is an innovative adjunct to existing immunotherapies
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