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When considering both main and interaction effects together, lnKabsC (GI absorption of iHg into the liver) and lnKunabsC (fecal elimination of unabsorbed iHg) generally have a greater impact on model simulations of AUC curves for blood and target tissue concentrations across species
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Although the action mechanism of SS-31 against kidney disease is not fully understood, the available studies suggest that it elicits its effects primarily by protecting mitochondrial structure, repairing damaged mitochondria, scavenging ROS, and increasing ATP supply, thereby reducing oxidative stress and improving apoptosis, inflammatory response, autophagy, and fibrosis [14, 27, 34] (Figure 1)
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