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The peptide appears to promote tenocyte proliferation and enhance extracellular matrix synthesis, leading to improved biomechanical properties of repaired tissues

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However, while most evidence suggests that copper-activated autophagy protects against cell death, some copper compounds such as copper oxide nanoparticles and Cu(II) complexes have been shown to induce autophagy-dependent cell death in cancer cells, which can be ameliorated by autophagy inhibitors [199,200]