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From a therapeutic perspective, the central role of GPX4 is not equivalent to the only critical node

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Synovitis—the swelling of the synovial membrane—serves as the front line, where fibroblasts and macrophages swell the ranks, releasing a cytokine storm, in RA, TNF-α, IL-6, and harvesting oxidative stress ‘characters’ all work together as partners in crime to compound and aggravate the situation on the cellular level, as well as at the patient level [5]