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For example, LPS- or TNF-α stimulates macrophages and microglia to induce robust neurotoxicity, but the destruction was completely blocked by MK-801 (dizoclipine), an NMDAR blocker.[279] Further, they demonstrated that blocking glutaminase, the enzyme that converts glutamine into glutamate [Figure 7], also blocked TNF-α and LPS neurotoxicity, as did blocking gap junctions, a principle exchange system for glutamate release

Tertiary: L-Arginin

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