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The interaction between macrophages and GSH relates both to the function of GSH as the main antioxidant of ROS and its role of signaling within the innate immune system [36]

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Transcriptomic analyses following short-term exposure to cisplatin or EGFR-targeted therapies revealed that early DTP cells rapidly acquire non-proliferative, stress-adaptive transcriptional programs, including alterations in chromatin-associated regulators, metabolic pathways, and survival-related genes

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